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1
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- Dizziness and balance disorders center
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2
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- Go into some basic principles
- applications and testing
- get into a few prominent diagnoses
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3
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- EIGHT MILLION PHYSICIAN VISITS/YR
- AVERAGE: 5 VISITS WITHOUT RESOLUTION OF PROBLEM
- LOSS OF LIVLIHOOD, FALLS INJURIES
- SYSTEMATIC APPROACH
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4
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- VERTIGO
- LIGHT-HEADEDNESS
- DYSEQUALIBRIUM
- GAIT DYSFUNCTION
- NEAR SYNCOPE
- ANXIETY
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5
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- A MULTIDIMENSIONAL APPROACH
- AREAS OF EXPERTISE
- NEUROLOGIST
- OTOLOGIST
- REHAB SPECIALIST
- SHUNTING
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6
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7
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8
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9
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10
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11
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- BPPV
- Meniere’s
- Vestibular neuritis
- Bilateral vestibular Loss
- Post-traumatic vertigo (labyrinthine concussion)
- Perilymph fistula
- Migraine and epilepsy
- Cerebro-vascular Disease
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12
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- Orthostatics and both arms
- Hallpike
- Fukada
- Head Thrust
- Head Shake
- Romberg (conventional, tandem, foam pad)
- Fistula test
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13
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- Recurrent
- One ear down position
- Positive Hallpike
- Transitory positional vertigo
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14
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- Posterior nystagmus are delayed
by approximately 15 seconds (latency), peak in 20-30 seconds, and then
decay, with complete resolution of symptoms.
- Symptoms and reversed nystagmus
may recur when the patient is brought to a sitting position.Nystagmus
fatigues on repeated trials. Peripheral nystagmus is latent, paroxysmal,
geotropic, reversible, and fatigable.
- Horizontal canal BPPV nystagmus is purely horizontal and asymmetric,
with its stronger component beating toward the diseased canal.
- Anterior canal nystagmus is
rotary, with its vertical component beating downward. The vertical
component of benign paroxysmal positioning nystagmus (BPPN) is best
observed by asking the patient to move the eyes away from the down-most
(tested) ear.
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15
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16
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17
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18
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- Form of Panic Attack
- Sensory overload
- “Supermarket Syndrome
- Sensory Overload
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19
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- Severe vertigo and vomiting
- Hearing Loss
- Fullness
- unilateral Tinnitus
- Endolymphatic Hydrops
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20
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21
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22
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- Compensations
- Avoidance (BPPV)
- Substitution (Bilateral Vestibular Loss)
- Plasticity (Vestibular Neuritis)
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23
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- HABITUATION
- ADAPTATION OF OTHER SENSORY SYSTEMS
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24
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- MS
- PD
- Aging
- Multi-sensory Deficit
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25
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26
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- May present as typical peripheral vestibulpathy
- ? lesion at root entry zone
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27
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- Aging
- Loss of neurons in CNS
- Arthritis
- Peripheral nerve dysfunction
- Vestibular dysfunction
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28
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- Physical therapy
- falls prevention
- muscle strengthening
- trying out assistive devices
- minimizing deficits
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29
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30
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- Unilateral Hearing Loss
- VII and V
- Unsteadiness rarely paroxysmal vertigo
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31
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- Vertigo
- Diplopia
- Dysarthria
- Dysphagia
- Ataxia
- Sensory or Motor Loss
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32
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- BPPV
- Meniere’s
- “Cervical” vertigo
- Perilymph fistula
- Factitious (psychological) vertigo
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33
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34
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- Hearing Loss
- Vertigo
- Bilateral “meniere’s”
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35
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- Anti HSP-70
- Anti Raji Cell
- Sed, ANA, RF, C1Q, FTA, Lyme, Thyroids
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36
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- Traumatic Communication betw inner and middle ear
- Pressure effect
- Tullio: vertigo and nystagmus to loud sound
- strain or blowing nose
- Fistula test
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37
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- Oscillopsia
- Visual Dependence
- Aminoglycosides
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38
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- CHRONIC SENSITIVITY TO MOTION
- OTHER PERSON DRIVING
- DISCOMFORT WITH MOTION
- VESTIBULAR REHAB: HABITUATION
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39
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- Persistence of perception of motion after a cruise
- Psychophysiological (?)
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